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As someone whose heart was damaged by Rheumatic Fever, this looks fairly similar between what we’re seeing with Covid and Covid Vaccinations (heart inflammation)- immune response going after the body’s own proteins:

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1905712/



Just in case anyone is concerned by the risk vaccination poses. The European Medical Agency recently reported that

* Pfizer-BioNTech - 145 cases of myocarditis and 138 cases of pericarditis out of 177m doses given

* Moderna - 19 case of myocarditis and 19 cases of pericarditis out of 20 million doses given

Five people died. The review said they were all either elderly or had other health conditions.


According to the CDC (as of August 18th) there are at least 742 cases of myocarditis and myopericarditis associated with vaccination, potentially upwards of ~1,300 [1].

For males age 16-17 the reporting rate of myopericarditis after 2 doses of Pfizer is 71.5 per 1 million doses administered (0.0071%) [1].

For males aged 18-24 the reporting rate after 2 doses of Pfizer or Moderna is ~37 per 1 million doses (0.0037%).

[1] https://www.cdc.gov/vaccines/acip/meetings/downloads/slides-...


Is slide #7 showing 20 times higher myopericarditis than baseline for 18-24 year old males?


Are these numbers higher or lower than what we’d expect to see in terms of myocarditis and myopericarditis cases resulting from infection with COVID-19?

I’m genuinely curious as I haven’t been able to find a good comparison on my own but I have read that damage to heart tissue is a potential long-term complication resulting from natural infection.


Great question, I'm only aware of one paper that somewhat answers that question in a limited sub-population (college athletes). To truly answer that question we would need perform cardiac magnetic resonance imaging on a large population-representative sample. If anyone is aware of such a study please do share.

> Representing 13 universities, cardiovascular testing was performed in 1597 athletes (964 men [60.4%]). Thirty-seven (including 27 men) were diagnosed with COVID-19 myocarditis (overall 2.3%; range per program, 0%-7.6%); 9 had clinical myocarditis and 28 had subclinical myocarditis. If cardiac testing was based on cardiac symptoms alone, only 5 athletes would have been detected (detected prevalence, 0.31%). Cardiac magnetic resonance imaging for all athletes yielded a 7.4-fold increase in detection of myocarditis (clinical and subclinical). Follow-up CMR imaging performed in 27 (73.0%) demonstrated resolution of T2 elevation in all (100%) and late gadolinium enhancement in 11 (40.7%).

[1] Prevalence of Clinical and Subclinical Myocarditis in Competitive Athletes With Recent SARS-CoV-2 Infection https://jamanetwork.com/journals/jamacardiology/fullarticle/...


That remind me of why antibiotics are used to treat Strep-Throat/Scarlet Fever. The antibiotics do not make a significant impact in the time it takes for one to recover from the bacteria. If I recall correctly, I think antibiotics speed up recovery by a day or two (someone correct me if I am wrong).

However, antibiotics are used to help prevent one from getting Rheumatic Fever from the bacteria.




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