That article smells fishy as hell. And if Alzheimer's were treatable, today, it would be a huge huge deal, not something being peddled around as a miracle cure on the basis of a 10-person "study".
They're making progress though. They've found at least one candidate drug that appears to clear up the Amyloid-beta plaques in mice.
One of the things I noticed right away is that the therapeutic programs prescribed to the patients patients in the study combined autophagy/ketosis (fasting 12 hours at night), low-glycemic diets, daily exercise, stress reduction, and sleep optimization.
A recent paper [1] concluded that "[...] the term 'type 3 diabetes' accurately reflects the fact that AD represents a form of diabetes that selectively involves the brain and has molecular and biochemical features that overlap with both type 1 diabetes mellitus and T2DM."
Something to think about.
[1] De la Monte SM, Wands JR. Alzheimer’s Disease Is Type 3 Diabetes–Evidence Reviewed. Journal of diabetes science and technology (Online). 2008;2(6):1101-1113. Link: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2769828/
If you inject amyloid-beta peptide extract from an AD-infected mouse into the brain of another, it then starts growing the usual plaques[1]. And A-beta plaque deposits are basically the distinguishing characteristic of Alzheimer's. So in my book that's very strong experimental evidence that A-beta is the causal mechanism of Alzheimer's and not merely a side effect.
It seems to be basically a misfolded protein, like CJD or mad-cow.
Usual caveats - nothing in science is ever proven to 100% certainty. Remove seal before use. Ask your doctor if Alzheimer's is right for you.
